R-plasmid transfer and its response to nalidixic acid
نویسندگان
چکیده
منابع مشابه
R-plasmid transfer and its response to nalidixic acid.
The conjugational transfer efficiency of 41 wild-type R-plasmids was studied in Escherichia coli K-12. Type I R-plasmids were transferred at comparatively high and rather uniform frequencies, whereas type F R-plasmids showed less uniform and, on average, somewhat lower transfer frequencies. R-plasmids not mediating sensitivity to F-, I-, or N-specific phages showed moderate transfer frequencies...
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Desdanine inhibited the plaque formation of male-specific coliphages but not that of other coliphages tested. Desdanine also suppressed the multiplication of both RNA phage Q beta and filamentous DNA phage f1 at the concentration of 3.13 approximately 6.25 mug/ml which had no influence on the growth of their host cells. However, the inhibitory effect on the phage multiplication was not due to t...
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15 صفحه اولPlasmid RP4 specifies a deoxyribonucleic acid primase involved in its conjugal transfer and maintenance.
We surveyed plasmids representative of most incompatibility groups for their conferred deoxyribonucleic acid (DNA) primase activity. RP4 (IncP) was one of the few with such activity although, unlike the derepressed IncIalpha plasmids (which also specify a primase), it did not suppress the dnaG mutation. Using deletion and Tn7 derivatives of RP4, we located the presumed primase structural gene (...
متن کاملPlasmid-mediated quinolone resistance in nalidixic-acid-susceptible strains of Salmonella enterica isolated in Scotland.
Sir, Chromosomal mutations in the topoisomerase II gene gyrA and altered drug accumulation were considered to be the only mechanisms of quinolone resistance until 1998, when the plasmidmediated quinolone resistance gene, qnrA, was described in an isolate of Klebsiella pneumoniae. Since then, other plasmidborne resistance genes, qnrB, qnrS (and their variants), aac(60)-Ib-cr and qepA, have been ...
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ژورنال
عنوان ژورنال: Journal of Bacteriology
سال: 1977
ISSN: 0021-9193,1098-5530
DOI: 10.1128/jb.131.1.76-81.1977